Joana Do Vale Viegas Silva Carvalho

Lab: Experimental Histopathology
Job Title: Senior Laboratory Research Scientist

Displaying 1 - 6 of 6

Last updated : 16 July 2020 03:09

The transcription co-repressors MTG8 and MTG16 regulate exit of intestinal stem cells from their niche and differentiation into enterocyte vs secretory lineages

Authors (11)

Gastroenterology

Published 15 June 2020

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γδ T cells compose a developmentally regulated intrauterine population and protect against vaginal candidiasis

Authors (18)

L Monin
DS Ushakov
H Arnesen
N Bah
A Jandke
M Muñoz-Ruiz
[...]
M Curtis
H Carlsen
U Steinhoff
P Boysen
Adrian Hayday

Mucosal Immunology

Published 29 May 2020

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Selective inhibition of cancer cell self-renewal through a Quisinostat-histone H1.0 axis

Authors (17)

Nature Communications

Published 14 April 2020

Crick labs/ facilities

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CD9 identifies pancreatic cancer stem cells and modulates glutamine metabolism to fuel tumour growth

Authors (14)

Nature Cell Biology

Published 4 November 2019

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RAC1P29S induces a mesenchymal phenotypic switch via serum response factor to promote melanoma development and therapy resistance

Authors (17)

Daniël A Lionarons
David Hancock
Sareena Rana
Philip East
Christopher Moore
Miguel Murillo
[...]
Ian Rosewell
Ralph Fritsch
Richard Neubig
Miriam Molina Arcas
Julian Downward

Cancer Cell

Published 8 July 2019

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SH3BP4 regulates intestinal stem cells and tumorigenesis by modulating β-catenin nuclear localization

Authors (9)

Cell Reports

Published 26 February 2019

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Joana Do Vale Viegas Silva Carvalho

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The transcription co-repressors MTG8 and MTG16 regulate exit of intestinal stem cells from their niche and differentiation into enterocyte vs secretory lineages

γδ T cells compose a developmentally regulated intrauterine population and protect against vaginal candidiasis

Selective inhibition of cancer cell self-renewal through a Quisinostat-histone H1.0 axis

CD9 identifies pancreatic cancer stem cells and modulates glutamine metabolism to fuel tumour growth

RAC1P29S induces a mesenchymal phenotypic switch via serum response factor to promote melanoma development and therapy resistance

SH3BP4 regulates intestinal stem cells and tumorigenesis by modulating β-catenin nuclear localization

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