Targeting adhesion to the vascular niche to improve therapy for acute myeloid leukemia
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Myriam LR Haltalli Cristina Lo CelsoAbstract
Niche hijack by malignant cells is considered to be a prominent cause of disease relapse. Barbier and colleagues uncover (E)-selectin as a novel mediator of malignant cell survival and regeneration which, upon blockade, has the potential to significantly improve therapeutic outcomes.
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Publisher website (DOI) 10.1038/s41467-020-17594-7
Europe PubMed Central 32703951
Pubmed 32703951
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